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Helminthiasis
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This article is about the infection. For the organisms, see Helminths.
Helminthiasis
Synonyms
helminthosis, helminthiases, helminth infection
Ascaris worms (one type of helminth) in the small bowel of an infected person in South Africa (X-ray image with barium as contrast medium)
Pronunciation
/ˌhɛlminˈθaɪəsis/
Classification and external resources
Specialty
infectious disease
ICD-10
B83.9
ICD-9-CM
128.9
DiseasesDB
28826
MeSH
D006373
[edit on Wikidata]
Helminthiasis (plural helminthiases), also known as worm infection, is any macroparasitic disease of humans and other animals in which a part of the body is infected with parasitic worms, known as helminths. There are numerous species of these parasites, which are broadly classified into tapeworms, flukes, and roundworms. They often live in the gastrointestinal tract of their hosts, but they may also burrow into other organs, where they induce physiological damage.
Soil-transmitted helminthiasis and schistosomiasis are the most important helminthiases, and are among the neglected tropical diseases.[1] This group of helmianthiases have been targeted under the joint action of the world's leading pharmaceutical companies and non-governmental organizations through a project launched in 2012 called the London Declaration on Neglected Tropical Diseases, which aims to control or eradicate certain neglected tropical diseases by 2020.[2]
Helminthiasis has been found to result in poor birth outcome, poor cognitive development, poor school and work performance, poor socioeconomic development, and poverty.[3][4] Chronic illness, malnutrition, and anemia are further examples of secondary effects.[5]
Soil-transmitted helminthiases are responsible for parasitic infections in as much as a quarter of the human population worldwide.[6] One well-known example of soil-transmitted helminthiases is ascariasis.
Contents
[hide]
1 Signs and symptoms
1.1 Mass and volume
1.2 Immunological response
2 Secondary effects
2.1 Immune changes
2.2 Chronic illness
2.3 Malnutrition
2.4 Anemia
2.5 Cognitive changes
3 Helminths types causing infections
3.1 Roundworm infection (nematodiasis)
3.2 Tapeworm infection (cestodiasis)
3.3 Trematode infection (trematodiasis)
3.4 Acanthocephala infection
4 Transmission
5 Diagnosis
6 Prevention
7 Treatment
7.1 Medications
7.2 Mass deworming
7.3 Surgery
8 Epidemiology
8.1 Neglected tropical diseases
8.2 Prevalence
8.3 Variations within communities
8.4 Disability-adjusted life years
8.5 Deaths
9 See also
10 References
11 External links
Signs and symptoms[edit]
Example clinical photo: Guinea worm infection (dracunculiasis), worm coming out of the foot of an infected person.
Ascaris infection: Antimesenteric splitting of the outer layers of the bowel wall due to a large amount of ascaris (South Africa)
The signs and symptoms of helminthiasis depend on a number of factors including: the site of the infestation within the body; the type of worm involved; the number of worms and their volume; the type of damage the infesting worms cause; and, the immunological response of the body. Where the burden of parasites in the body is light, there may be no symptoms.
Certain worms may cause particular constellations of symptoms. For instance, taeniasis can lead to seizures due to neurocysticercosis.[7]
Mass and volume[edit]
In extreme cases of intestinal infestation, the mass and volume of the worms may cause the outer layers of the intestinal wall, such as the muscular layer, to tear. This may lead to peritonitis, volvulus, and gangrene of the intestine.[8]
Immunological response[edit]
As pathogens in the body, helminths induce an immune response. Immune-mediated inflammatory changes occur in the skin, lung, liver, intestine, central nervous system, and eyes. Signs of the body's immune response may include eosinophilia, edema, and arthritis.[9] An example of the immune response is the hypersensitivity reaction that may lead to anaphylaxis. Another example is the migration of Ascaris larvae through the bronchi of the lungs causing asthma.[10]
Secondary effects[edit]
Immune changes[edit]
See also: Effects of parasitic worms on the immune system
Micrograph of cut section of human appendix showing a pin worm.
In humans, T helper cells and eosinophils respond to helminth infestation. Inflammation leads to encapsulation of egg deposits throughout the body. Helminths excrete into the intestine toxic substances after they feed. These substances then enter the circulatory and lymphatic systems of the host body.
Chronic immune responses to helminthiasis may lead to increased susceptibility to other infections such as tuberculosis, HIV, and malaria.[11][12][13] There is conflicting information about whether deworming reduces HIV progression and viral load and increases CD4 counts in antiretroviral naive and experienced individuals.[14][15]
Chronic illness[edit]
Chronic helminthiasis may cause severe morbidity.[16] Helminthiasis has been found to result in poor birth outcome, poor cognitive development, poor school and work performance, decreased productivity, poor socioeconomic development, and poverty.[3][4][5]
Malnutrition[edit]
Helminthiasis may cause chronic illness through malnutrition including vitamin deficiencies, stunted growth, anemia, and protein-energy malnutrition. Worms compete directly with their hosts for nutrients, but the magnitude of this effect is likely minimal as the nutritional requirements of worms is relatively small.[17][18][19] In pigs and humans, Ascaris has been linked to lactose intolerance and vitamin A, amino acid, and fat malabsorption.[3] Impaired nutrient uptake may result from direct damage to the intestinal mucosal wall or from more subtle changes such as chemical imbalances and changes in gut flora.[20] Alternatively, the worms’ release of protease inhibitors to defend against the body’s digestive processes may impair the breakdown of other nutrients.[17][19] In addition, worm induced diarrhoea may shorten gut transit time, thus reducing absorption of nutrients.[3]
Malnutrition due to worms can give rise to anorexia.[18] A study of 459 children in Zanzibar revealed spontaneous increases in appetite after deworming.[21] Anorexia might be a result of the body’s immune response and the stress of combating infection.[19] Specifically, some of the cytokines released in the immune response to worm infestation have been linked to anorexia in animals.[17]
Anemia[edit]
Helminths may cause iron-deficiency anemia. This is most severe in heavy hookworm infections, as Necator americanus and Ancylostoma duodenale feed directly on the blood of their hosts. Although the daily consumption of an individual worm (0.02–0.07 ml and 0.14–0.26 ml respectively) is small, the collective consumption under heavy infection can be clinically significantly.[3][19] Intestinal whipworm may also cause anemia. Anemia has also been associated with reduced stamina for physical labor, a decline in the ability to learn new information, and apathy, irritability, and fatigue.[3] A study of the effect of deworming and iron supplementation in 47 students from the Democratic Republic of the Congo found that the intervention improved cognitive function.[22] Another study found that in 159 Jamaican schoolchildren, deworming led to better auditory short-term memory and scanning and retrieval of long-term memory over a period of nine-weeks.[23]
Cognitive changes[edit]
Malnutrition due to helminths may affect cognitive function leading to low educational performance, decreased concentration and difficulty with abstract cognitive tasks. Iron deficiency in infants and preschoolers is associated with "lower scores ... on tests of mental and motor development ... [as well as] increased fearfulness, inattentiveness, and decreased social responsiveness".[17] Studies in the Philippines and Indonesia found a significant correlation between helminthiasis and decreased memory and fluency.[24][25] Large parasite burdens, particularly severe hookworm infections, are also associated with absenteeism, under-enrollment, and attrition in school children.[17]
Helminths types causing infections[edit]
Ascaris life cycle: Adult worms in the lumen of the small intestine (1). The female produces eggs (approximately 200,000 per day) that are excreted with the feces (2). Unfertilized eggs are harmless, but fertilized ones are infective after 18 days to several weeks (3). Infective eggs are ingested (4), enter the gut (5), develop into larvae in the intestine, and penetrate the blood vessel to enter lungs, where they develop further (6), after 10 to 14 days, penetrate the alveolar walls, ascend the bronchial tree to the throat, and are re-swallowed (7). Upon reaching the small intestine, they develop into adult worms (8). It takes 2 to 3 months for one complete cycle. Adult worms can live 1 to 2 years.
Collage of various helminth eggs, from left to right: Trichosomoides egg, Ascaris lumbricoides with larva hatching, sample of adult roundworms, Hymenolepis nana, Schistosoma mansoni and Toxocara canis with larva hatching
Of all the known helminth species, the most important helminths with respect to understanding their transmission pathways, their control, inactivation and enumeration in samples of human excreta from dried feces, faecal sludge, wastewater, and sewage sludge are:[26]
soil-transmitted helminths, including Ascaris lumbricoides (the most common worldwide), Trichuris trichiura, Necator americanus, Strongyloides stercoralis and Ancylostoma duodenale
Hymenolepis nana
Taenia saginata
Enterobius
Fasciola hepatica
Schistosoma mansoni
Toxocara canis
Toxocara cati
Helminthiases are classified as follows (the disease names end with "-sis" and the causative worms are in brackets):
Roundworm infection (nematodiasis)[edit]
Filariasis (Wuchereria bancrofti, Brugia malayi infection)
Onchocerciasis (Onchocerca volvulus infection)
Soil-transmitted helminthiasis – this includes ascariasis (Ascaris lumbricoides infection, trichuriasis (Trichuris infection), and hookworm infection (includes necatoriasis and Ancylostoma duodenale infection)
Trichostrongyliasis (Trichostrongylus spp. infection)
Dracunculiasis (guinea worm infection)
Baylisascaris (raccoon roundworm, may be transmitted to pets livestock and humans)
Tapeworm infection (cestodiasis)[edit]
Echinococcosis (Echinococcus infection)
Hymenolepiasis (Hymenolepis infection)
Taeniasis/cysticercosis (Taenia infection)
Coenurosis (T. multiceps, T. serialis, T. glomerata, and T. brauni infection)
Trematode infection (trematodiasis)[edit]
Amphistomiasis (amphistomes infection)
Clonorchiasis (Clonorchis sinensis infection)
Fascioliasis (Fasciola infection)
Fasciolopsiasis (Fasciolopsis buski infection)
Opisthorchiasis (Opisthorchis infection)
Paragonimiasis (Paragonimus infection)
Schistosomiasis/bilharziasis (Schistosoma infection)
Acanthocephala infection[edit]
Moniliformis infection
Transmission[edit]
Helminths are transmitted to the final host in several ways. The most common infection is through ingestion of contaminated vegetables, drinking water, and raw or undercooked meat. Contaminated food may contain eggs of nematodes such as Ascaris, Enterobius, and Trichuris; cestodes such as Taenia, Hymenolepis, and Echinococcus; and treamtodes such as Fasciola. Raw or undercooked meats are the major sources of Taenia (pork, beef and venison), Trichinella (pork and bear), Diphyllobothrium (fish), Clonorchis (fish), and Paragonimus (crustaceans). Schistosomes and nematodes such as hookworms (Ancylostoma and Necator) and Strongyloides can penetrate the skin directly. Finally, Wuchereria, Onchocerca, and Dracunculus are transmitted by mosquitoes and flies.[16] In the developing world, the use of contaminated water is a major risk factor for infection.[27] Infection can also take place through the practice of geophagy, which is not uncommon in parts of sub-Saharan Africa. Soil is eaten, for example, by children or pregnant women to counteract a real or perceived deficiency of minerals in their diet.[28]
Diagnosis[edit]
Identification and quantification of helminth eggs at UNAM university in Mexico City, Mexico
Specific helminths can be identified through microscopic examination of their eggs (ova) found in faecal samples. The number of eggs is measured in units of eggs per gram.[29] However, it does not quantify mixed infections, and in practice, is inaccurate for quantifying the eggs of schistosomes and soil-transmitted helminths.[30] Sophisticated tests such as serological assays, antigen tests, and molecular diagnosis are also available;[29][31] however, they are time-consuming, expensive and not always reliable.[32]
Prevention[edit]
Disrupting the cycle of the worm will prevent infestation and re-infestation. Prevention of infection can largely be achieved by addressing the issues of WASH—water, sanitation and hygiene.[33][34][35] The reduction of open defecation is particularly called for,[36][37] as is stopping the use of human waste as fertilizer.[6]
Further preventive measures include adherence to appropriate food hygiene, wearing of shoes, regular deworming of pets, and the proper disposal of their feces.[3]
Scientists are also searching for a vaccine against helminths, such as a hookworm vaccine.[38]
Treatment[edit]
Medications[edit]
Main article: Anthelmintic
Broad-spectrum benzimidazoles (such as albendazole and mebendazole) are the first line treatment of intestinal roundworm and tapeworm infections. Macrocyclic lactones (such as ivermectin) are effective against adult and migrating larval stages of nematodes. Praziquantel is the drug of choice for schistosomiasis, taeniasis, and most types of food-borne trematodiases. Oxamniquine is also widely used in mass deworming programmes. Pyrantel is commonly used for veterinary nematodiasis.[39][40] Artemisinins and derivatives are proving to be candidates as drugs of choice for trematodiasis.[41]
Mass deworming[edit]
Main article: Mass deworming
In regions where helminthiasis is common, mass deworming treatments may be performed, particularly among school-age children, who are a high-risk group.[42][43] Most of these initiatives are undertaken by the World Health Organization (WHO) with positive outcomes in many regions.[44][45] Deworming programs can improve school attendance by 25 percent.[46] Although deworming improves the health of an individual, outcomes from mass deworming campaigns, such as reduced deaths or increases in cognitive ability, nutritional benefits, physical growth, and performance, are uncertain or not apparent.[47][48][49][50]
Surgery[edit]
Example of ascariasis (ascaris infection) - Difficult surgical procedure in South Africa on a gangrenous piece of bowel that had to be cut out; live ascaris worms are emerging.
Piece of intestine, blocked by worms, surgically removed from a 3-year-old boy in South Africa.[51]
If complications of helminthiasis, such as intestinal obstruction occur, emergency surgery may be required.[8][51] Patients who require non-emergency surgery, for instance for removal of worms from the biliary tree, can be pre-treated with the anthelmintic drug albendazole.[8]
Epidemiology[edit]
Areas with the highest prevalence of helminthiasis are tropical and subtropical areas including sub-Saharan Africa, central and east Asia, and the Americas.
Neglected tropical diseases[edit]
Some types of helminthiases are classified as neglected tropical diseases.[1][52] They include:
Soil-transmitted helminthiases
Roundworm infections such as lymphatic filariasis, dracunculiasis, and onchocerciasis
Trematode infections, such as schistosomiasis, and food-borne trematodiases, including fascioliasis, clonorchiasis, opisthorchiasis, and paragonimiasis
Tapeworm infections such as cysticercosis, taeniasis, and echinococcosis
Prevalence[edit]
The soil-transmitted helminths (A. lumbricoides, T. trichiura, N. americanus, A. duodenale), schistosomes, and filarial worms collectively infect more than a quarter of the human population worldwide at any one time, far surpassing HIV and malaria together.[29][31] Schistosomiasis is the second most prevalent parasitic disease of humans after malaria.[53]
In 2014–15, the WHO estimated that approximately 2 billion people were infected with soil-transmitted helminthiases,[6] 249 million with schistosomiasis,[54] 56 million people with food-borne trematodiasis,[55] 120 million with lymphatic filariasis,[56] 37 million people with onchocerciasis,[57] and 1 million people with echinococcosis.[58] Another source estimated a much higher figure of 3.5 billion infected with one or more soil-transmitted helminths.[59][60]
In 2014, only 148 people were reported to have dracunculiasis because of a successful eradication campaign for that particular helminth, which is easier to eradicate than other helminths as it is transmitted only by drinking contaminated water.[61]
Because of their high mobility and lower standards of hygiene, school-age children are particularly vulnerable to helminthiasis.[62] Most children from developing nations will have at least one infestation. Multi-species infections are very common.[63]
Variations within communities[edit]
Even in areas of high prevalence, the frequency and severity of infection is not uniform within communities or families.[64] A small proportion of community members harbour the majority of worms, and this depends on age. The maximum worm burden is at five to ten years of age, declining rapidly thereafter.[65] Individual predisposition to helminthiasis for people with the same sanitation infrastructure and hygiene behavior is thought to result from differing immunocompetence, nutritional status, and genetic factors.[64] Because individuals are predisposed to a high or a low worm burden, the burden reacquired after successful treatment is proportional to that before treatment.[64]
Disability-adjusted life years[edit]
It is estimated that intestinal nematode infections cause 5 million disability-adjusted life years (DALYS) to be lost, of which hookworm infections account for more than 3 million DALYS and ascaris infections more than 1 million.[66] There are also signs of progress: The Global Burden of Disease Study published in 2015 estimates a 46 percent (59 percent when age standardised) reduction in years lived with disability (YLD) for the 13-year time period from 1990 to 2013 for all intestinal/nematode infections, and even a 74 percent (80 percent when age standardised) reduction in YLD from ascariasis.[67]
Deaths[edit]
As many as 135,000 die annually from soil transmitted helminthiasis.[3][68][69]
The 1990–2013 Global Burden of Disease Study estimated 5,500 direct deaths from schistosomiasis,[70] while more than 200,000 people were estimated in 2013 to die annually from causes related to schistosomiasis.[71] Another 20 million have severe consequences from the disease.[72] It is the most deadly of the neglected tropical diseases.[73]
Helminth genera
Common name
Infections (million per year)
Direct deaths per year
Regions where common
Soil transmitted helminthiasis (STH) (classified as neglected tropical disease):
Ascaris lumbricoides Roundworm 1000 to 1450
807 to 1,121[74]
20,000 Many regions of South-east Asia, Africa, and Central and South America[75][76][77][78][79][80]
Trichuris trichiura Whipworm 500
604–795[74]
In moist, warm, tropical regions of Asia, Africa, Central and South America, and the Caribbean islands.[77][78][79][80][81]
Ancylostoma duodenale Hookworm 900 to 1300
576–740 (hookworm in general)[82]
In tropical and subtropical countries (Sub-Saharan Africa)[78][81]
Necator americanus
Strongyloides stercoralis Hookworm, pinworm 50 to 100 Thousands In moist rainy areas of the tropics and subtropics, in some areas of southern and eastern Europe and of the United States of America[78][79]
All STH together 1500 to 2000[6] 135,000[3][68][69] Tropical and subtropical areas, in particular sub-Saharan Africa, the Americas, China and east Asia.[6]
Not transmitted via soil but classified as neglected tropical disease:
Schistosoma mansoni Blood fluke All types of Schistosoma together: 160 to 200
(210 "affected"[83])
12,000[84] 150,000 deaths from renal failure[85]
200,000 indirect deaths from "causes related to" Schistosomiasis[71]
In tropical and subtropical regions[77][78][79][80][81]
Schistosoma haematobium 112 (in Sub-Saharan Africa alone)[85]
Echinococcus granulosus 3[86] Developing countries
Not transmitted via soil and not classified as neglected tropical disease:
Toxocara canis Dog roundworm 50 Many regions of South-east Asia, Africa, and Central and South America[75][76][77][78][79][80]
Taenia solium Pork tapeworm 50 South America, Southeast Asia, West Africa and East Africa[77][78][79][80]
Taenia saginata Beef tapeworm 50
(all types of Taenia: 40 to 60[87])
Hymenolepis nana Dwarf tapeworm 100
Hymenolepis diminuta Rat tapeworm
Fasciola hepatica,
Fascioloides magna Liver fluke 50 Largely in southern and eastern Asia but also in central and eastern Europe[78][79]
Fasciolopsis buski Giant intestinal fluke
Dracunculus medinensis Guinea worm Nowadays negligible thanks to eradication program[88] Formerly widespread in India, west Africa and southern Sudan[78][79]
Trichostrongylus orientalis Roundworm 1–3 ("several") Rural communities in Asia[78][79]
Other 100 Worldwide[78][79]
Total (number of infections) Approx. 3.5 billion Worldwide
See also[edit]
Kato technique
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External links[edit]
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text:
2018 World Men's Curling Championship
From Wikipedia, the free encyclopedia
Jump to: navigation, search
2018 World Men's Curling Championship
Host city
Las Vegas, Nevada
Arena
Orleans Arena
Dates
March 31 – April 8, 2018
Attendance
74,829[1]
Winner
Sweden
Curling club
Karlstads CK, Karlstad
Skip
Niklas Edin
Third
Oskar Eriksson
Second
Rasmus Wranå
Lead
Christoffer Sundgren
Alternate
Henrik Leek
Finalist
Canada (Brad Gushue)
« 2017
2019 »
The 2018 World Men's Curling Championship (branded as the 361˚ World Men's Curling Championship 2018 for sponsorship reasons) was held from March 31 to April 8, 2018 at Orleans Arena, on the Las Vegas Strip in Paradise, Nevada, United States.
In a rematch of the 2017 gold medal game, the Swedish team led by Niklas Edin beat the defending champion Canadian team led by Brad Gushue to win the championship.
Contents
[hide]
1 Qualification
2 Teams
3 Round robin standings
3.1 WCT ranking
4 Round robin results
4.1 Draw 1
4.2 Draw 2
4.3 Draw 3
4.4 Draw 4
4.5 Draw 5
4.6 Draw 6
4.7 Draw 7
4.8 Draw 8
4.9 Draw 9
4.10 Draw 10
4.11 Draw 11
4.12 Draw 12
4.13 Draw 13
4.14 Draw 14
4.15 Draw 15
4.16 Draw 16
4.17 Draw 17
4.18 Draw 18
4.19 Draw 19
4.20 Draw 20
5 Playoffs
5.1 Qualification Games
5.2 Semifinal 1
5.3 Semifinal 2
5.4 Bronze Medal Game
5.5 Gold Medal Game
6 Statistics
6.1 Top 5 Player percentages
6.2 Perfect games
7 References
8 External links
Qualification[edit]
The following nations are qualified to participate in the 2018 World Men's Curling Championship:
United States (host country)
One team from the Americas zone
Canada (winner of the 2018 Americas Challenge)
Eight teams from the 2017 European Curling Championships
Germany
Italy
Netherlands
Norway
Russia
Scotland
Sweden
Switzerland
Three teams from the 2017 Pacific-Asia Curling Championships
China
Japan
South Korea
Teams[edit]
Canada
China
Germany[2]
Italy[3]
Japan
Bally Haly G&CC &
St. John's CC, St. John's
Skip: Brad Gushue
Third: Mark Nichols
Second: Brett Gallant
Lead: Geoff Walker
Alternate: Thomas Sallows
Harbin CC, Harbin
Skip: Zou Dejia
Third: Zou Qiang
Second: Xu Jingtao
Lead: Shao Zhilin
Alternate: Ma Yanlong
Baden Hills G&CC, Rheinmünster
Skip: Alexander Baumann
Third: Manuel Walter
Second: Daniel Herberg
Lead: Sebastian Schweizer
Alternate: Ryan Sherrard
A.S.D. Trentino Curling, Cembra
Fourth: Amos Mosaner
Skip: Joël Retornaz
Second: Andrea Pilzer
Lead: Daniele Ferrazza
Alternate: Fabio Ribotta
Sapporo CA, Sapporo
Fourth: Go Aoki
Skip: Masaki Iwai
Second: Ryotaro Shukuya
Lead: Yutaka Aoyama
Alternate: Koji Nisato
Netherlands[4]
Norway[5]
Russia
Scotland
South Korea
CC PWA Zoetermeer, Zoetermeer
Skip: Jaap van Dorp
Third: Wouter Gösgens
Second: Laurens Hoekman
Lead: Carlo Glasbergen
Alternate: Alexander Magan
Oppdal CC, Oppdal
Skip: Steffen Walstad
Third: Markus Høiberg
Second: Magnus Nedregotten
Lead: Magnus Vågberg
Alternate: Steffen Mellemseter
Adamant CC, Saint Petersburg
Skip: Alexey Timofeev
Third: Sergey Glukhov
Second: Artur Razhabov
Lead: Evgeny Klimov
Alternate: Artem Shmakov
Gogar Park CC, Edinburgh
Skip: Bruce Mouat
Third: Grant Hardie
Second: Bobby Lammie
Lead: Hammy McMillan Jr.
Alternate: Ross Paterson
Uiseong CC, Uiseong
Skip: Kim Chang-min
Third: Seong Se-hyeon
Second: Oh Eun-su
Lead: Lee Ki-bok
Alternate: Kim Min-chan
Sweden[6]
Switzerland
United States
Karlstads CK, Karlstad
Skip: Niklas Edin
Third: Oskar Eriksson
Second: Rasmus Wranå
Lead: Christoffer Sundgren
Alternate: Henrik Leek
CC Bern, Bern
Skip: Marc Pfister
Third: Enrico Pfister
Second: Raphael Märki
Lead: Simon Gempeler
Alternate: Jan Hess
St. Paul CC, St. Paul
Fourth: Greg Persinger
Skip: Rich Ruohonen
Second: Colin Hufman
Lead: Philip Tilker
Alternate: Christopher Plys
Round robin standings[edit]
Final Round Robin Standings
Key
Teams to Playoffs
Country
Skip
W
L
PF
PA
Ends Won
Ends Lost
Blank Ends
Stolen Ends
Shot Pct.
Sweden Niklas Edin 11 1 79 44 45 40 20 7 88%
Scotland Bruce Mouat 11 1 92 53 53 41 11 16 86%
Canada Brad Gushue 9 3 86 55 51 41 11 14 87%
South Korea Kim Chang-min 7 5 84 71 48 48 17 9 83%
Norway Steffen Walstad 7 5 78 72 53 44 15 16 86%
United States Rich Ruohonen 6 6 65 66 48 46 25 11 84%
Switzerland Marc Pfister 6 6 75 82 53 47 17 13 81%
Italy Joël Retornaz 5 7 79 76 47 48 15 9 81%
Russia Alexey Timofeev 5 7 71 85 48 51 16 12 80%
Netherlands Jaap van Dorp 4 8 66 77 46 55 17 5 81%
Japan Masaki Iwai 3 9 60 87 41 51 15 6 78%
China Zou Dejia 3 9 60 84 46 51 14 9 81%
Germany Alexander Baumann 1 11 53 96 41 57 11 4 78%
WCT ranking[edit]
Year to date World Curling Tour order of merit ranking for each team prior to the event.[7]
Nation (Skip)
Rank
Points
Sweden (Edin) 1 455.02
Canada (Gushue) 2 431.42
Scotland (Mouat) 7 285.29
South Korea (Kim) 13 238.06
Norway (Walstad) 16 200.67
United States (Ruohonen) 21 162.77
Switzerland (Pfister) 34 114.24
Germany (Baumann) 42 94.76
Italy (Retornaz) 49 84.00
Netherlands (van Dorp) 50 82.52
China (Zou) 86 45.28
Russia (Timofeev) 145 21.15
Japan (Iwai) 682 0.00
Round robin results[edit]
All draw times are listed in Pacific Daylight Time (UTC−7:00).[8]
Draw 1[edit]
Saturday, March 31, 13:30
Sheet A 1 2 3 4 5 6 7 8 9 10 Final
Sweden (Edin) 1 0 0 1 0 0 3 0 0 1 6
Germany (Baumann) 0 0 1 0 1 1 0 0 2 0 5
Sheet B 1 2 3 4 5 6 7 8 9 10 Final
Netherlands (van Dorp) 1 0 1 0 1 0 1 0 0 0 4
South Korea (Kim) 0 1 0 1 0 1 0 1 2 1 7
Sheet C 1 2 3 4 5 6 7 8 9 10 Final
China (Zou) 0 0 0 1 0 0 2 2 0 1 6
Switzerland (Pfister) 0 1 1 0 0 2 0 0 1 0 5
Sheet D 1 2 3 4 5 6 7 8 9 10 Final
Russia (Timofeev) 0 0 1 0 2 1 1 0 1 X 6
Canada (Gushue) 3 0 0 2 0 0 0 2 0 X 7
Draw 2[edit]
Saturday, March 31, 18:30
Sheet A 1 2 3 4 5 6 7 8 9 10 Final
Norway (Walstad) 0 2 0 0 0 3 1 4 X X 10
Italy (Retornaz) 0 0 1 0 3 0 0 0 X X 4
Sheet B 1 2 3 4 5 6 7 8 9 10 11 Final
United States (Ruohonen) 0 0 2 1 0 1 0 0 1 0 1 6
Japan (Iwai) 2 0 0 0 0 0 1 1 0 1 0 5
Sheet C 1 2 3 4 5 6 7 8 9 10 Final
Canada (Gushue) 2 0 2 0 1 0 0 1 1 0 7
Scotland (Mouat) 0 2 0 1 0 1 2 0 0 2 8
Sheet D 1 2 3 4 5 6 7 8 9 10 Final
Sweden (Edin) 0 2 0 2 0 3 0 0 X X 7
Switzerland (Pfister) 0 0 1 0 1 0 0 1 X X 3
Draw 3[edit]
Sunday, April 1, 08:30
Sheet A 1 2 3 4 5 6 7 8 9 10 11 Final
Russia (Timofeev) 0 1 0 0 2 0 0 2 1 0 1 7
China (Zou) 1 0 0 1 0 1 1 0 0 2 0 6
Sheet B 1 2 3 4 5 6 7 8 9 10 Final
Norway (Walstad) 2 2 0 1 2 0 1 X X X 8
Germany (Baumann) 0 0 1 0 0 1 0 X X X 2
Sheet C 1 2 3 4 5 6 7 8 9 10 Final
Japan (Iwai) 0 0 1 0 1 0 0 X X X 2
South Korea (Kim) 0 1 0 3 0 3 2 X X X 9
Sheet D 1 2 3 4 5 6 7 8 9 10 Final
United States (Ruohonen) 0 2 0 0 2 0 0 0 0 0 4
Netherlands (van Dorp) 2 0 0 1 0 0 0 1 1 1 6
Draw 4[edit]
Sunday, April 1, 13:30
Sheet A 1 2 3 4 5 6 7 8 9 10 Final
South Korea (Kim) 0 1 0 1 0 0 2 0 2 1 7
United States (Ruohonen) 0 0 1 0 3 0 0 1 0 0 5
Sheet B 1 2 3 4 5 6 7 8 9 10 Final
Scotland (Mouat) 0 0 0 1 2 0 0 3 0 0 6
Italy (Retornaz) 0 0 2 0 0 0 1 0 0 1 4
Sheet C 1 2 3 4 5 6 7 8 9 10 Final
Netherlands (van Dorp) 0 0 0 0 1 0 0 1 0 X 2
Sweden (Edin) 2 0 0 0 0 0 2 0 1 X 5
Sheet D 1 2 3 4 5 6 7 8 9 10 Final
Japan (Iwai) 0 0 1 0 2 0 1 0 0 X 4
Norway (Walstad) 2 0 0 1 0 2 0 2 1 X 8
Draw 5[edit]
Sunday, April 1, 18:30
Sheet A 1 2 3 4 5 6 7 8 9 10 Final
Scotland (Mouat) 1 0 2 1 0 1 0 1 0 0 6
Sweden (Edin) 0 1 0 0 3 0 2 0 0 1 7
Sheet B 1 2 3 4 5 6 7 8 9 10 Final
Canada (Gushue) 2 2 0 2 1 0 X X X X 7
Switzerland (Pfister) 0 0 1 0 0 1 X X X X 2
Sheet C 1 2 3 4 5 6 7 8 9 10 Final
Italy (Retornaz) 0 1 1 0 0 2 0 2 0 3 9
Russia (Timofeev) 2 0 0 2 0 0 1 0 1 0 6
Sheet D 1 2 3 4 5 6 7 8 9 10 Final
China (Zou) 0 0 2 0 1 0 1 0 2 0 6
Germany (Baumann) 0 1 0 0 0 1 0 2 0 1 5
Draw 6[edit]
Monday, April 2, 08:30
Sheet B 1 2 3 4 5 6 7 8 9 10 11 Final
Japan (Iwai) 1 2 0 0 2 0 0 0 0 1 1 7
Netherlands (van Dorp) 0 0 2 0 0 0 2 1 1 0 0 6
Sheet C 1 2 3 4 5 6 7 8 9 10 Final
Norway (Walstad) 2 0 0 0 1 0 1 0 1 2 7
United States (Ruohonen) 0 1 1 1 0 0 0 2 0 0 5
Draw 7[edit]
Monday, April 2, 13:30
Sheet A 1 2 3 4 5 6 7 8 9 10 Final
Italy (Retornaz) 0 1 0 2 0 3 0 1 0 0 7
Canada (Gushue) 2 0 2 0 1 0 1 0 1 1 8
Sheet B 1 2 3 4 5 6 7 8 9 10 Final
South Korea (Kim) 0 1 0 1 0 1 0 1 0 X 4
China (Zou) 3 0 2 0 2 0 1 0 1 X 9
Sheet C 1 2 3 4 5 6 7 8 9 10 Final
Switzerland (Pfister) 0 1 1 0 0 1 0 2 0 2 7
Germany (Baumann) 0 0 0 2 0 0 1 0 1 0 4
Sheet D 1 2 3 4 5 6 7 8 9 10 11 Final
Scotland (Mouat) 0 2 0 2 0 0 0 1 0 1 1 7
United States (Ruohonen) 1 0 2 0 0 2 0 0 1 0 0 6
Draw 8[edit]
Monday, April 2, 18:30
Sheet A 1 2 3 4 5 6 7 8 9 10 11 Final
Germany (Baumann) 0 0 1 0 1 0 2 0 3 0 1 8
Netherlands (van Dorp) 1 0 0 2 0 2 0 1 0 1 0 7
Sheet B 1 2 3 4 5 6 7 8 9 10 Final
Russia (Timofeev) 3 0 2 0 1 0 0 1 3 0 10
Norway (Walstad) 0 2 0 2 0 2 1 0 0 1 8
Sheet C 1 2 3 4 5 6 7 8 9 10 Final
China (Zou) 0 0 0 1 0 1 0 0 2 X 4
Japan (Iwai) 0 0 1 0 2 0 2 2 0 X 7
Sheet D 1 2 3 4 5 6 7 8 9 10 Final
South Korea (Kim) 1 0 1 0 0 0 1 0 1 0 4
Sweden (Edin) 0 1 0 1 0 1 0 1 0 1 5
Draw 9[edit]
Tuesday, April 3, 08:30
Sheet A 1 2 3 4 5 6 7 8 9 10 Final
Japan (Iwai) 0 2 0 2 0 0 0 1 0 0 5
Russia (Timofeev) 3 0 1 0 1 0 0 0 0 1 6
Sheet B 1 2 3 4 5 6 7 8 9 10 Final
Germany (Baumann) 0 1 0 0 1 1 1 0 X X 4
Scotland (Mouat) 1 0 3 2 0 0 0 3 X X 9
Sheet C 1 2 3 4 5 6 7 8 9 10 11 Final
South Korea (Kim) 0 0 0 0 2 0 0 2 0 2 0 6
Canada (Gushue) 0 1 0 2 0 2 0 0 1 0 1 7
Sheet D 1 2 3 4 5 6 7 8 9 10 Final
Switzerland (Pfister) 0 1 0 1 0 0 1 2 0 1 6
Italy (Retornaz) 1 0 1 0 0 2 0 0 1 0 5
Draw 10[edit]
Tuesday, April 3, 13:30
Sheet A 1 2 3 4 5 6 7 8 9 10 Final
China (Zou) 1 0 1 0 1 0 1 0 1 0 5
Norway (Walstad) 0 1 0 1 0 1 0 2 0 1 6
Sheet B 1 2 3 4 5 6 7 8 9 10 Final
United States (Ruohonen) 0 0 0 1 0 1 0 X X X 2
Sweden (Edin) 2 0 0 0 3 0 2 X X X 7
Sheet C 1 2 3 4 5 6 7 8 9 10 Final
Scotland (Mouat) 1 2 0 2 0 1 0 2 X X 8
Netherlands (van Dorp) 0 0 0 0 2 0 1 0 X X 3
Sheet D 1 2 3 4 5 6 7 8 9 10 Final
Canada (Gushue) 3 0 2 0 2 2 X X X X 9
Japan (Iwai) 0 1 0 1 0 0 X X X X 2
Draw 11[edit]
Tuesday, April 3, 18:30
Sheet A 1 2 3 4 5 6 7 8 9 10 Final
Switzerland (Pfister) 0 3 0 2 2 0 1 0 2 2 12
South Korea (Kim) 0 0 4 0 0 3 0 4 0 0 11
Sheet B 1 2 3 4 5 6 7 8 9 10 Final
China (Zou) 0 0 1 0 2 0 0 2 0 X 5
Italy (Retornaz) 2 1 0 1 0 0 2 0 5 X 11
Sheet C 1 2 3 4 5 6 7 8 9 10 Final
Sweden (Edin) 1 0 0 1 0 1 0 0 0 0 3
Norway (Walstad) 0 0 1 0 1 0 1 1 0 1 5
Sheet D 1 2 3 4 5 6 7 8 9 10 Final
Germany (Baumann) 1 0 0 0 2 0 2 0 1 0 6
Russia (Timofeev) 0 0 1 1 0 2 0 3 0 1 8
Draw 12[edit]
Wednesday, April 4, 08:30
Sheet A 1 2 3 4 5 6 7 8 9 10 Final
Sweden (Edin) 0 4 0 3 2 0 X X X X 9
Italy (Retornaz) 1 0 2 0 0 1 X X X X 4
Sheet B 1 2 3 4 5 6 7 8 9 10 Final
Norway (Walstad) 0 0 0 1 0 1 0 X X X 2
Canada (Gushue) 2 0 1 0 2 0 3 X X X 8
Sheet C 1 2 3 4 5 6 7 8 9 10 Final
United States (Ruohonen) 1 0 0 0 2 2 0 0 1 0 6
Russia (Timofeev) 0 0 2 2 0 0 1 0 0 2 7
Sheet D 1 2 3 4 5 6 7 8 9 10 Final
South Korea (Kim) 1 0 0 0 0 1 0 X X X 2
Scotland (Mouat) 0 0 2 1 0 0 4 X X X 7
Draw 13[edit]
Wednesday, April 4, 13:30
Sheet A 1 2 3 4 5 6 7 8 9 10 Final
United States (Ruohonen) 0 1 0 2 0 2 0 0 2 1 8
Germany (Baumann) 0 0 2 0 2 0 0 1 0 0 5
Sheet B 1 2 3 4 5 6 7 8 9 10 11 Final
Switzerland (Pfister) 2 0 0 2 0 1 0 0 0 1 1 7
Russia (Timofeev) 0 1 0 0 2 0 2 1 0 0 0 6
Sheet C 1 2 3 4 5 6 7 8 9 10 Final
Japan (Iwai) 0 2 0 2 0 1 0 1 X X 6
Italy (Retornaz) 4 0 3 0 1 0 2 0 X X 10
Sheet D 1 2 3 4 5 6 7 8 9 10 Final
Netherlands (van Dorp) 2 0 0 1 0 2 0 0 0 1 6
China (Zou) 0 2 0 0 1 0 1 1 0 0 5
Draw 14[edit]
Wednesday, April 4, 18:30
Sheet A 1 2 3 4 5 6 7 8 9 10 Final
Netherlands (van Dorp) 1 0 0 1 0 1 0 2 0 X 5
Canada (Gushue) 0 2 1 0 3 0 1 0 1 X 8
Sheet B 1 2 3 4 5 6 7 8 9 10 Final
Scotland (Mouat) 2 0 2 1 0 3 1 X X X 9
Japan (Iwai) 0 1 0 0 1 0 0 X X X 2
Sheet C 1 2 3 4 5 6 7 8 9 10 Final
Germany (Baumann) 0 1 0 1 0 1 2 0 1 0 6
South Korea (Kim) 2 0 1 0 3 0 0 1 0 3 10
Sheet D 1 2 3 4 5 6 7 8 9 10 11 Final
Norway (Walstad) 0 3 0 0 1 0 0 1 1 0 0 6
Switzerland (Pfister) 3 0 0 1 0 0 0 0 0 2 2 8
Draw 15[edit]
Thursday, April 5, 08:30
Sheet A 1 2 3 4 5 6 7 8 9 10 Final
Norway (Walstad) 1 0 0 0 1 0 2 0 2 0 6
Scotland (Mouat) 0 1 0 1 0 2 0 3 0 1 8
Sheet B 1 2 3 4 5 6 7 8 9 10 Final
Sweden (Edin) 4 3 1 1 0 0 1 X X X 10
China (Zou) 0 0 0 0 1 1 0 X X X 2
Sheet C 1 2 3 4 5 6 7 8 9 10 Final
Netherlands (van Dorp) 0 2 0 1 0 2 0 1 0 2 8
Switzerland (Pfister) 1 0 1 0 1 0 1 0 2 0 6
Sheet D 1 2 3 4 5 6 7 8 9 10 11 Final
Italy (Retornaz) 1 0 0 0 0 0 2 0 1 1 0 5
South Korea (Kim) 0 1 2 1 0 0 0 1 0 0 1 6
Draw 16[edit]
Thursday, April 5, 13:30
Sheet A 1 2 3 4 5 6 7 8 9 10 Final
Germany (Baumann) 0 1 0 0 1 0 0 1 0 X 3
Japan (Iwai) 1 0 0 2 0 3 1 0 1 X 8
Sheet B 1 2 3 4 5 6 7 8 9 10 Final
Italy (Retornaz) 0 0 1 0 0 1 0 1 0 0 3
United States (Ruohonen) 0 1 0 0 1 0 1 0 1 1 5
Sheet C 1 2 3 4 5 6 7 8 9 10 Final
Canada (Gushue) 0 0 1 0 3 0 0 3 0 X 7
China (Zou) 1 0 0 1 0 1 1 0 0 X 4
Sheet D 1 2 3 4 5 6 7 8 9 10 Final
Sweden (Edin) 2 1 0 0 0 2 0 0 2 X 7
Russia (Timofeev) 0 0 1 0 1 0 0 1 0 X 3
Draw 17[edit]
Thursday, April 5, 18:30
Sheet A 1 2 3 4 5 6 7 8 9 10 Final
Canada (Gushue) 0 2 0 0 1 0 0 0 2 0 5
United States (Ruohonen) 1 0 1 1 0 0 2 0 0 1 6
Sheet B 1 2 3 4 5 6 7 8 9 10 Final
Netherlands (van Dorp) 1 0 0 1 0 0 3 0 1 0 6
Norway (Walstad) 0 1 1 0 0 2 0 2 0 1 7
Sheet C 1 2 3 4 5 6 7 8 9 10 Final
Russia (Timofeev) 0 0 0 1 1 0 0 X X X 2
Scotland (Mouat) 1 1 1 0 0 2 2 X X X 7
Sheet D 1 2 3 4 5 6 7 8 9 10 11 Final
Switzerland (Pfister) 2 0 2 0 0 2 0 1 0 2 1 10
Japan (Iwai) 0 1 0 2 0 0 3 0 3 0 0 9
Draw 18[edit]
Friday, April 6, 08:30
Sheet A 1 2 3 4 5 6 7 8 9 10 Final
Scotland (Mouat) 1 0 0 3 0 0 1 0 2 1 8
Switzerland (Pfister) 0 2 1 0 1 1 0 1 0 0 6
Sheet B 1 2 3 4 5 6 7 8 9 10 Final
Canada (Gushue) 0 1 0 0 0 1 2 0 1 0 5
Sweden (Edin) 3 0 0 1 0 0 0 1 0 1 6
Sheet C 1 2 3 4 5 6 7 8 9 10 Final
Italy (Retornaz) 0 2 0 1 0 3 3 2 X X 11
Germany (Baumann) 1 0 2 0 1 0 0 0 X X 4
Sheet D 1 2 3 4 5 6 7 8 9 10 Final
United States (Ruohonen) 1 1 0 3 0 0 0 1 0 1 7
China (Zou) 0 0 1 0 0 1 0 0 2 0 4
Draw 19[edit]
Friday, April 6, 13:30
Sheet A 1 2 3 4 5 6 7 8 9 10 Final
Japan (Iwai) 0 1 0 1 0 0 0 1 0 X 3
Sweden (Edin) 1 0 2 0 0 0 3 0 1 X 7
Sheet B 1 2 3 4 5 6 7 8 9 10 Final
China (Zou) 0 1 0 1 1 1 0 X X X 4
Scotland (Mouat) 6 0 1 0 0 0 2 X X X 9
Sheet C 1 2 3 4 5 6 7 8 9 10 Final
South Korea (Kim) 0 0 0 2 0 0 0 4 0 3 9
Norway (Walstad) 0 1 1 0 0 2 0 0 1 0 5
Sheet D 1 2 3 4 5 6 7 8 9 10 Final
Russia (Timofeev) 0 0 1 0 1 1 2 1 0 0 6
Netherlands (van Dorp) 2 2 0 1 0 0 0 0 0 2 7
Draw 20[edit]
Friday, April 6, 18:30
Sheet A 1 2 3 4 5 6 7 8 9 10 Final
Italy (Retornaz) 0 0 0 1 0 1 1 2 0 1 6
Netherlands (van Dorp) 0 0 1 0 3 0 0 0 1 0 5
Sheet B 1 2 3 4 5 6 7 8 9 10 Final
Russia (Timofeev) 0 0 0 2 0 2 0 0 X X 4
South Korea (Kim) 0 1 1 0 4 0 0 3 X X 9
Sheet C 1 2 3 4 5 6 7 8 9 10 Final
Switzerland (Pfister) 0 0 1 0 0 1 0 0 0 1 3
United States (Ruohonen) 0 2 0 0 1 0 0 1 1 0 5
Sheet D 1 2 3 4 5 6 7 8 9 10 Final
Germany (Baumann) 0 0 0 0 1 0 X X X X 1
Canada (Gushue) 1 1 1 2 0 3 X X X X 8
Playoffs[edit]
Qualification Games Semifinals Gold Medal Game
1 Sweden 9
4 South Korea 7 4 South Korea 8
5 Norway 5 1 Sweden 7
3 Canada 3
2 Scotland 5
3 Canada 6 3 Canada 9
6 United States 4
Bronze Medal Game
2 Scotland 11
4 South Korea 4
Qualification Games[edit]
Saturday, April 7, 08:30
Sheet B 1 2 3 4 5 6 7 8 9 10 Final
South Korea (Kim) 2 0 1 0 0 1 0 2 0 1 7
Norway (Walstad) 0 1 0 2 0 0 1 0 1 0 5
Player percentages
South Korea
Norway
Lee Ki-bok 84% Magnus Vågberg 81%
Oh Eun-su 83% Magnus Nedregotten 83%
Seong Se-hyeon 83% Markus Høiberg 73%
Kim Chang-min 85% Steffen Walstad 80%
Total 84% Total 79%
Sheet D 1 2 3 4 5 6 7 8 9 10 Final
Canada (Gushue) 1 1 0 0 0 1 0 2 0 1 6
United States (Ruohonen) 0 0 0 1 0 0 2 0 1 0 4
Player percentages
Canada
United States
Geoff Walker 88% Philip Tilker 80%
Brett Gallant 73% Colin Hufman 79%
Mark Nichols 93% Rich Ruohonen 85%
Brad Gushue 91% Greg Persinger 81%
Total 86% Total 81%
Semifinal 1[edit]
Saturday, April 7, 13:30
Sheet C 1 2 3 4 5 6 7 8 9 10 11 Final
Sweden (Edin) 1 0 0 3 0 0 2 0 2 0 1 9
South Korea (Kim) 0 1 1 0 2 2 0 1 0 1 0 8
Player percentages
Sweden
South Korea
Christoffer Sundgren 76% Lee Ki-bok 87%
Rasmus Wranå 84% Oh Eun-su 83%
Oskar Eriksson 97% Seong Se-hyeon 75%
Niklas Edin 83% Kim Chang-min 70%
Total 85% Total 79%
Semifinal 2[edit]
Saturday, April 7, 18:30
Sheet C 1 2 3 4 5 6 7 8 9 10 Final
Scotland (Mouat) 2 0 0 2 0 1 0 0 0 X 5
Canada (Gushue) 0 2 1 0 1 0 3 1 1 X 9
Player percentages
Scotland
Canada
Hammy McMillan Jr. 92% Geoff Walker 79%
Bobby Lammie 82% Brett Gallant 88%
Grant Hardie 86% Mark Nichols 82%
Bruce Mouat 65% Brad Gushue 94%
Total 81% Total 86%
Bronze Medal Game[edit]
Sunday, April 8, 12:00
Team 1 2 3 4 5 6 7 8 9 10 Final
Scotland (Mouat) 1 0 3 2 4 0 1 0 X X 11
South Korea (Kim) 0 2 0 0 0 1 0 1 X X 4
Player percentages
Scotland
South Korea
Hammy McMillan Jr. 71% Lee Ki-bok 100%
Bobby Lammie 97% Oh Eun-su 85%
Grant Hardie 89% Seong Se-hyeon 65%
Bruce Mouat 92% Kim Chang-min 72%
Total 87% Total 80%
Gold Medal Game[edit]
Sunday, April 8, 17:00
Team 1 2 3 4 5 6 7 8 9 10 Final
Sweden (Edin) 0 0 2 2 1 0 2 0 X X 7
Canada (Gushue) 0 0 0 0 0 1 0 2 X X 3
Player percentages
Sweden
Canada
Christoffer Sundgren 88% Geoff Walker 88%
Rasmus Wranå 92% Brett Gallant 94%
Oskar Eriksson 88% Mark Nichols 91%
Niklas Edin 95% Brad Gushue 80%
Total 91% Total 88%
Statistics[edit]
Top 5 Player percentages[edit]
Round robin only[9]
Leads
%
Geoff Walker 91
Hammy McMillan Jr. 91
Christoffer Sundgren 90
Magnus Vågberg 90
Lee Ki-bok 89
Seconds
%
Bobby Lammie 88
Magnus Nedregotten 88
Rasmus Wranå 87
Brett Gallant 86
Oh Eun-su 85
Thirds
%
Mark Nichols 87
Oskar Eriksson 86
Rich Ruohonen 86
Markus Høiberg 85
Grant Hardie 84
Skips
%
Niklas Edin 90
Brad Gushue 85
Greg Persinger 83
Bruce Mouat 82
Steffen Walstad 82
Perfect games[edit]
Player
Team
Position
Opponent
Lee Ki-bok South Korea Lead China
South Korea Lead Scotland
Evgeny Klimov Russia Lead Sweden
Evgeny Klimov Russia Lead South Korea
Hammy McMillan Jr. Scotland Lead China
Shao Zhilin China Lead Scotland
Daniel Herberg Germany Second Scotland
Magnus Nedregotten Norway Second Scotland
Seong Se-hyeon South Korea Third Russia
References[edit]
Jump up ^ "Sweden's Edin wins world curling championship with 7-3 win over Canada's Gushue". Vancourier.com. 2018-04-08. Retrieved 2018-04-22.
Jump up ^ http://www.curling-verband.de/news/3avvkx.html
Jump up ^ "dettaglio-squadra-naz - FISG - Federazione Italiana Sport del Ghiaccio". Fisg.it. Retrieved 2018-04-22.
Jump up ^ "Nederland Wint Goud op WCT Moscow Classic (nieuws) - Nederlandse Curling Bond". Curling.nl. Retrieved 2018-04-22.
Jump up ^ "Flamboyant Ulsrud back on the medal trail - Olympic News". Olympic.org. 2018-02-04. Retrieved 2018-04-22.
Jump up ^ https://www.lasvegascurling.rocks/gold-rush-continues
Jump up ^ http://www.worldcurl.com/oom/oom.php?task=week&orderby=ytd&oomid=82&eventyear=2017&week=31
Jump up ^ "Official Draw". World Curling Federation. Retrieved February 5, 2018.
Jump up ^ http://odf2.worldcurling.co/data/CUR_WMCC2018P/Men's_Teams/C84C~Shot_Success_Percentage_by_Position.pdf
External links[edit]
"2018 World Men's Curling Championship". World Curling Federation.
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